Aprender pela experimentacao (isto é , na pratica) é mais rapido.
Faz o teste toma o bicarbonato ou faz o teste numa chavena: bicarbonato com vinagre ou com limao. Ve a reacao. O que ves é dioxido de carbono, daí o arrotanço.
Ae puseres bicarbonato numa substancia nao acida, nao tens reacao ou meste caso efervescencia
a reacção é NaHCO3 - + HCl -> H2O(l) + CO2(g) + NaCl (aq)
A reacção com o limao é uma reacção diferente (o ácido citrico e o ácido cloridrico não são o mesmo ácido), mas na prática a reacção é semelhante (forma-se um sal (citrato de sódio), água e dioxido de carbono.
O arrotar ou não, depende de pessoa para pessoa (da própria fisiologia da pessoa), e da quantidade de ácido que têm (que depende de coisas tão simples como se estão a comer ou não). O dizer que se arrota é pq tem muito ácido e não arrota é porque tem pouco, é um pouco ignorar restantes factores.
A quantidade de CO2 que se liberta dependerá da quantidade de bicarbonato que ingerires. De qualquer das formas o "arrotar" é uma fraca forma de o avaliar.
Quanto ao resto, ainda aguardo a explicação de a azia ser devida a pouco ácido...
Costumas ter azia?
https://www.amazon.com/Why-Stomach-Acid-Good-You/dp/0871319314 Jonathan wright, le o livro
Dr. Jonathan Wright is the Medical Director of Tahoma Clinic where he also practices medicine. A Harvard University (A.B. 1965) and University of Michigan graduate (M.D. 1969), he continues to be a forerunner in research and application of natural treatments for healthy aging and illness. Along with Alan Gaby, M.D., he has since 1976 accumulated a file of over 50,000 research papers about diet, vitamins, minerals, botanicals, and other natural substances from which he has developed non-patent medicine (non-“drug”) treatments for health problems. Dr. Wright has taught natu
Causes & Effects of Hypochlorhydria
The underlying pathologies of insufficient acid secretion are varied and at times unclear. Achlorhydria is an expression of atrophic gastritis, which is associated with an increased risk of gastric cancer.4 Ruling out gastric carcinoma is important early in assessment, due to its aggressive nature and 5-year survival rate of only 20%.5 Autoimmune atrophic gastritis and pernicious anemia results from autoantibodies against the parietal cell and intrinsic factor, preventing the absorption of B12.6 Acute Helicobacter pylori (H pylori) infection is a cause of transient hypochlorhydria,7 which occurs to facilitate the survival and colonization of H pylori.8
Most chronic infections with H pylori lead to pangastritis and decreased acid production.3 In children with H pylori infection, low stomach acid has been shown to reduce serum iron and transferrin, playing a role in refractory iron deficiency anemia.9 It has been hypothesized that low stomach acid reduces protein absorption and is a risk factor for depression, due to the relatively reduced levels of tryptophan and tyrosine in the brain.10 A low level of pepsin with hypochlorhydria causes incomplete protein digestion, which may lead to improper absorption of macromolecules into the circulation, as well as immune-complex deposition diseases.11 Other causes of hypochlorhydria include chronic overeating, hypoadrenalism, chronic stress, excess intake of processed foods and carbohydrates, caffeine, and alcohol.12
Diagnosis of Low Stomach Acid
The rationale for assessment of gastric acid production is usually considered during the history portion of a patient intake. Reported symptoms often relate to indigestion or functional dyspepsia developing during or after meals.13 Complaints such as gas, bloating, muscle cramps, epigastric heaviness and easy satiety can relate to a hypochlorhydric state, although the condition can also be asymptomatic.13 The symptomatic state is related to several factors, including mucosal integrity, parietal cell concentration, extrinsic inhibition, gastric reserve, and/or lower esophageal sphincter tone, to highlight a few theoretical causes.
Clinical presentation can vary widely, and hypochlorhydria should always be considered in patients with deficient states of depleted micro- and macromolecules, low vitality, and changes in gastrointestinal function. Signs may include deficiencies of vitamins and minerals, muscle cramps and twitches, acne, food allergies, dilated capillaries on cheeks and nose, brittle and peeling nails, and halitosis.11,13 Neurological symptoms due to nerve demyelination, including paresthesias or numbness and gait problems, are related to vitamin B12 deficiency caused by inadequate intrinsic factor from parietal cell loss. If pernicious anemia is suspected, testing for anti-parietal cell and anti-intrinsic factor antibodies is warranted, as the Schilling test is no longer used.14 To evaluate vitamin B12 deficiency, serum homocysteine and methylmalonic acid are most useful in making the diagnosis.14 Although not gastric-specific, a complete digestive stool analysis may be useful in assessing the nature of a gastrointestinal illness and involved organs.
The Heidelberg test, involving a capsule containing a pH-sensitive radio-transmitting device, has been in use since the 1960s.15 A small pH sensor, with or without a tether, is swallowed, and gastric acid fluctuation is evaluated before and after ingestion of a sodium bicarbonate solution. The presence of a tether allows the pH of gastric contents to be measured and evaluated through challenge; the absence of a tether allows the capsule to flow through, giving information on emptying time and intestinal pH.16 This method only provides a measure of pH, with no information on gastric acid quantity.15 Using the Heidelberg capsule allows categorizing the degree of hypochlorhydria with rate-of-return to baseline pH after a challenge, further narrowing the best treatment options for the observed condition.17
https://nutritionreview.org/2013/04/gastric-balance-heartburn-caused-excess-acid/
